منابع مشابه
P3: Neuronal Death Following Posttraumatic Excitability and Seizure
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متن کاملNeuronal Cell Death
penheim, 1991; Burek and Oppenheim, 1998). Although Neurons may die at many different developmental stages some of these hypotheses have received experimental and for many different reasons. As in other tissues, difsupport in individual systems, none seems to be generferent stimuli produce quite different morphological ally applicable. Indeed, it remains to be demonstrated that manifestations o...
متن کاملp3: neuronal death following posttraumatic excitability and seizure
seizure may occur after mild traumatic brain injury (tbi), and the severity of tbi can be considered the most crucial factor for an increased risk of recurring seizures as well as for the development of posttraumatic epilepsy. however, the effect of seizures in epileptogenesis after mild tbi cannot yet be accurately confirmed. this study was designed to determine whether mild tbi increases seiz...
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182 J Clin Psychiatry 58:5, May 1997 ying is generally an undesired affair. In recent years, however, it has become clear that many cells, including neurons, are actively programmed to commit molecular hari-kari. Such a process is called apoptosis. This is not the messy affair associated with cellular poisoning or suffocation, known as necrosis. Necrotic cell death is characterized by a severe ...
متن کاملNeuronal injury and death following focal mild brain injury: The role of network excitability and seizure
Objective(s): While traumatic brain injury (TBI) is a predisposing factor for development of post-traumatic epilepsy (PTE), the occurrence of seizures following brain trauma can infuriate adverse consequences of brain injury. However, the effect of seizures in epileptogenesis after mild TBI cannot yet be accurately confirmed. This study was designed to investigate the ...
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ژورنال
عنوان ژورنال: Journal of Cell Biology
سال: 2006
ISSN: 1540-8140,0021-9525
DOI: 10.1083/jcb.1724fta1